Please use this identifier to cite or link to this item: http://dspace.hmtu.edu.vn/handle/DHKTYTHD_123/406
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dc.contributor.authorManwani, Deepa-
dc.contributor.authorFrenette, Paul S.-
dc.date.accessioned2015-11-30T08:50:10Z-
dc.date.available2015-11-30T08:50:10Z-
dc.date.issued2013-
dc.identifier.urihttp://220.231.117.85:8000/handle/DHKTYTHD_123/406-
dc.description.abstractRecurrent and unpredictable episodes of vaso-occlusion are the hallmark of sickle cell disease. Symptomatic management and prevention of these events using the fetal hemoglobin–reactivating agent hydroxyurea are currently the mainstay of treatment. Discoveries over the past 2 decades have highlighted the important contributions of various cellular and soluble participants in the vaso-occlusive cascade. The role of these elements and the opportunities for therapeutic intervention are summarized in this review.vi
dc.language.isoenvi
dc.publisherAmerican Society of Hematologyvi
dc.titleVaso-occlusion in sickle cell disease: pathophysiology and novel targeted therapiesvi
dc.typeArticlevi
Appears in CollectionsHuyết học = Hematology

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